What were the Big Ideas in the reading and the class discussion in week #7?
ES 34
Ch. 12
This chapter describes the body’s immune response to the bacteria associated with gingivitis and periodontitis. Because of the microscopic reactions of the body through innate immune responses and specific immune responses, we are able to see some of the effects of these mechanisms clinically, such as erythema and edema.
Ch. 13
When microorganisms are able to evade the host’s ability to destroy bacteria it aids in the progression of periodontal disease. Depending on the host’s immune reaction to these microorganisms and the type of microorganisms that are present, this helps determine how severe the periodontitis will be.
The Big idea for chapter 12. In this chapter author focuses on role of immune system in inflammation. There two type of immune response: innate response which is early defense mechanism that involved in fighting invading microorganisms; adaptive immune responce activated later in the disease process and characterized by production of specific antibodies.
The big idea for chapter 13.
The author explains the interaction of microorganisms with host in periodontal diseases. The author describes the virulence factors that help microorganisms initiate the disease process. YB #23
The big idea's in chapter 12: What cells are involved in immunity and inflammation. The immune response consists of innate immune responses (first defense) and specific immune responses (protecting against).
The big idea's in chapter 13: Bacterial pathogens along with host tissues cause periodontal disease.
Ch.12 Big idea was about immunity and inflamation and how the innate or adaptive immune responce are triggerd by gingivitis and or periodontitis.
Ch.13 Big idea was about how microbs interact with the host in periodontal diseases.
Ch. 12
The immune system is classified under two different categories: innate and adaptive. These immune responses respond differently in the presence of pathogens. The cells involved in the inflammation process respond in the presence of gingivitis and periodontitis. When tissues become damaged there are cells that have different functions that help fight against pathogens.
Ch. 13
Describes how bacterial microorganisms affect the host response during periodontal disease. There are several different types of periodontal diseases that have different characteristics; some more severe than others, and some progress at different rates.
Chp. 12: Role of the immune system in inflammation. Includes the host response to bacterial invasion by first initiating early phase defense cells which is the innate immunity response and then the specific immune response protects the host against specific pathogens by the development of antibodies.
Chp. 13: Microbial interactions with the host and the ability of the microbe to cause disease; virulence factor. Adherence and bacterial evasion represent virulence factors for periodontal disease because it leads to colonization. Tissue destruction results from the interaction of bacteria or bacterial substances with host cells, which lead to degradation of the periodontal tissues. Different bacteria are present in the many types of periodontitis that exist.
ACS #35
Ch. 12: The body has the innate immune response which activates cells, such as macrophages and neutrophils, that can kill many types of organisms. There is also the specific immune response which activates T and B lymphocytes that can form memories of pathogens already encountered and kill those on the second exposure. Both types of the immune responses prevent infection and respond to inflammation from foreign pathogens.
Ch. 13: There are many types of oral bacteria but not all are considered pathogenic. The pathogenic organisms that cause periodontitis have the specific ability to harm the host but resist the immune response. This is called virulence. The level of virulence of an organism can vary, but in order to be pathogenic, the organism must be able to colonize the host tissues and cause tissue destruction. Bacteria have different means of doing this such as adhesion properties, release of endotoxins, etc.
Chapter 12- discusses the immune response and how the inflammatory response responds in the presence of gingivitis and periodontitis.
Chapter 13- discusses how bacteria affect the host response during periodontal disease
Ch.12 Big idea is how the immune system plays an important role in the inflammatory process. When the tissue is altered by specific bacteria and or trauma cells of the immune system either be innate or adaptive are activated to respond to the site.
Ch.13 Specific bacterial pathogens seems to be responsible for the disease process and depending on the host tissue the specific bacteria will respond differently and either slowly or rapidly destruct the tissue.
Ch. 12
Gingivitis and periodontitis are inflammatory responses and Ch. 12 discusses the role of the immune system in inflammation. Innate immunity provides a critical early-phase defense against invading microorganisms. Adaptive immunity or specific immune response provides the host with the ability to recognize specific pathogens and allows the immune system to respond more rapidly and effectively.
Ch. 13
Discusses the interaction of bacterial microorganisms and how they affect the host, which determines the course and extent of the resulting disease.
Chapter 12- The big idea is how the immune system is related to periodontal disease. The immune system has two groups innate and adaptive which involves many cells.
Chapter 13- Along with specific microorganisms the host defense plays a key role in destruction of tissues.
Ch. 12- This chapter talks about the immune response and all the different types of cells that are associated with it, and how they play a role in this response.
Ch. 13- This chapter is about the host defense and how it effects the oral tissues. It also talks about that everyone has different bacterial plaque which is why some people have certain changes in their gingival tissue, while others might have a different bacterial plaque, causing a different change in the tissue.
SF 25
---
C12-13
The big idea is that the body is equipped with defenses that stop infections. The innate response do not distinguish between different antigens and the acquired are defenses that build over time. infections began as acute infections if the body cannot fight them off it then enters the chronic inflammation period which can cause destruction to host tissues. In periodontitis the destruction of host tissue involves a constant battle between the host defenses (good soldiers) and bacterial defenses such as LPS (bad soldiers), and the alveolar bone is a casualty of the war.
Ch. 12- The big idea is the relationship between the immune system in inflammation and periodontal disease and the immune systems role. The chapter explains all the different cells that are involved with the immune system and inflammation,and also the two different immune response such as innate and adaptive.
CH. 13- The big idea is that different microbes interact with the host in periodontal disease. Specific bacterial pathogens appear to be responsible for the changes resulting in disease.
AS36
Chapter 12:
Discusses how the host response plays a role in the pathogenesis of periodontal disease. Innate immunity defends against invading microorganisms in the early phase and adaptive immunity helps the host become more efficient in protecting against pathogens.
Chapter 13:
Discusses how the pathogenesis of periodontal disease depends on the pathogens ability to destroy the tissue and the host tissues.
What were the Big Ideas in the reading and the class discussion in week #7?
ES 34
Ch. 12
This chapter describes the body’s immune response to the bacteria associated with gingivitis and periodontitis. Because of the microscopic reactions of the body through innate immune responses and specific immune responses, we are able to see some of the effects of these mechanisms clinically, such as erythema and edema.
Ch. 13
When microorganisms are able to evade the host’s ability to destroy bacteria it aids in the progression of periodontal disease. Depending on the host’s immune reaction to these microorganisms and the type of microorganisms that are present, this helps determine how severe the periodontitis will be.
The Big idea for chapter 12. In this chapter author focuses on role of immune system in inflammation. There two type of immune response: innate response which is early defense mechanism that involved in fighting invading microorganisms; adaptive immune responce activated later in the disease process and characterized by production of specific antibodies.
The big idea for chapter 13.
The author explains the interaction of microorganisms with host in periodontal diseases. The author describes the virulence factors that help microorganisms initiate the disease process. YB #23
The big idea's in chapter 12: What cells are involved in immunity and inflammation. The immune response consists of innate immune responses (first defense) and specific immune responses (protecting against).
The big idea's in chapter 13: Bacterial pathogens along with host tissues cause periodontal disease.
Ch.12 Big idea was about immunity and inflamation and how the innate or adaptive immune responce are triggerd by gingivitis and or periodontitis.
Ch.13 Big idea was about how microbs interact with the host in periodontal diseases.
Ch. 12
The immune system is classified under two different categories: innate and adaptive. These immune responses respond differently in the presence of pathogens. The cells involved in the inflammation process respond in the presence of gingivitis and periodontitis. When tissues become damaged there are cells that have different functions that help fight against pathogens.
Ch. 13
Describes how bacterial microorganisms affect the host response during periodontal disease. There are several different types of periodontal diseases that have different characteristics; some more severe than others, and some progress at different rates.
Chp. 12: Role of the immune system in inflammation. Includes the host response to bacterial invasion by first initiating early phase defense cells which is the innate immunity response and then the specific immune response protects the host against specific pathogens by the development of antibodies.
Chp. 13: Microbial interactions with the host and the ability of the microbe to cause disease; virulence factor. Adherence and bacterial evasion represent virulence factors for periodontal disease because it leads to colonization. Tissue destruction results from the interaction of bacteria or bacterial substances with host cells, which lead to degradation of the periodontal tissues. Different bacteria are present in the many types of periodontitis that exist.
ACS #35
Ch. 12: The body has the innate immune response which activates cells, such as macrophages and neutrophils, that can kill many types of organisms. There is also the specific immune response which activates T and B lymphocytes that can form memories of pathogens already encountered and kill those on the second exposure. Both types of the immune responses prevent infection and respond to inflammation from foreign pathogens.
Ch. 13: There are many types of oral bacteria but not all are considered pathogenic. The pathogenic organisms that cause periodontitis have the specific ability to harm the host but resist the immune response. This is called virulence. The level of virulence of an organism can vary, but in order to be pathogenic, the organism must be able to colonize the host tissues and cause tissue destruction. Bacteria have different means of doing this such as adhesion properties, release of endotoxins, etc.
Chapter 12- discusses the immune response and how the inflammatory response responds in the presence of gingivitis and periodontitis.
Chapter 13- discusses how bacteria affect the host response during periodontal disease
Ch.12 Big idea is how the immune system plays an important role in the inflammatory process. When the tissue is altered by specific bacteria and or trauma cells of the immune system either be innate or adaptive are activated to respond to the site.
Ch.13 Specific bacterial pathogens seems to be responsible for the disease process and depending on the host tissue the specific bacteria will respond differently and either slowly or rapidly destruct the tissue.
Ch. 12
- Gingivitis and periodontitis are inflammatory responses and Ch. 12 discusses the role of the immune system in inflammation. Innate immunity provides a critical early-phase defense against invading microorganisms. Adaptive immunity or specific immune response provides the host with the ability to recognize specific pathogens and allows the immune system to respond more rapidly and effectively.
Ch. 13Chapter 12- The big idea is how the immune system is related to periodontal disease. The immune system has two groups innate and adaptive which involves many cells.
Chapter 13- Along with specific microorganisms the host defense plays a key role in destruction of tissues.
Ch. 12- This chapter talks about the immune response and all the different types of cells that are associated with it, and how they play a role in this response.
Ch. 13- This chapter is about the host defense and how it effects the oral tissues. It also talks about that everyone has different bacterial plaque which is why some people have certain changes in their gingival tissue, while others might have a different bacterial plaque, causing a different change in the tissue.
SF 25
---
C12-13
The big idea is that the body is equipped with defenses that stop infections. The innate response do not distinguish between different antigens and the acquired are defenses that build over time. infections began as acute infections if the body cannot fight them off it then enters the chronic inflammation period which can cause destruction to host tissues. In periodontitis the destruction of host tissue involves a constant battle between the host defenses (good soldiers) and bacterial defenses such as LPS (bad soldiers), and the alveolar bone is a casualty of the war.
Ch. 12- The big idea is the relationship between the immune system in inflammation and periodontal disease and the immune systems role. The chapter explains all the different cells that are involved with the immune system and inflammation,and also the two different immune response such as innate and adaptive.
CH. 13- The big idea is that different microbes interact with the host in periodontal disease. Specific bacterial pathogens appear to be responsible for the changes resulting in disease.
AS36
Chapter 12:
Discusses how the host response plays a role in the pathogenesis of periodontal disease. Innate immunity defends against invading microorganisms in the early phase and adaptive immunity helps the host become more efficient in protecting against pathogens.
Chapter 13:
Discusses how the pathogenesis of periodontal disease depends on the pathogens ability to destroy the tissue and the host tissues.